视觉发育

  • 网络Visual development;Vision Development
视觉发育视觉发育
  1. 黑鲷视觉发育与摄食的关系

    The relation between feeding and vision development of black porgy

  2. 越来越多的证据表明神经营养家族参与神经发育的可塑性,并对视觉发育起着至关重要的作用。

    More and more evidences indicated that neurotrophins could participate in the plasticity of neural development , and play an essential role in vision development .

  3. 结论:屈光参差性弱视眼的P和M通道可能都受到了视觉发育敏感期异常视觉经验的影响;

    Conclusions : It was infered that abnormal visual experiences during early development may affect either P pathway or M pathway in anisometropic amblyopia .

  4. 小鼠孕期哺乳期补充DHA对仔鼠视觉发育影响的研究

    Influence of Supplementary DHA in Pregnancy and Lactation mice on the Visual Development of Pups

  5. NMDA受体在视觉发育过程中的作用

    Effect of NMDA receptor in visual development

  6. GABA及受体参与视觉发育可塑性关键期终止机制研究

    Study on Mechanisms by Which GABA Transmitter and Recepter Terminate the Critical Period of Rat Visual Development

  7. F-VEP对大鼠视觉发育阶段的判断具有一定价值。

    F-VEP is the valuable index for estimating the visual developing stages of rats .

  8. 深入研究NMDA受体在视觉发育过程中的作用,对于儿童弱视的治疗具有非常重要的临床意义。

    So , further study on effect of NMDA receptors on visual development has a great clinical significance for therapy of strabismus and amblyopia of children .

  9. 目的了解在视觉发育可塑期,NMDA-R1受体基因的mRNA构型与斜视性弱视猫视皮层神经元细胞水平表达减少有关的。

    Purpose To identify the expression of alternatively spliced mRNA isoforms of the NMDA R1 in the visual cortex of strabismic cats .

  10. LBA对剥夺性弱视大鼠视觉发育的保护机制可能与上调Bcl-2及NGF的表达有关,以促进神经细胞的营养和生长。

    The neuroprotective mechanism of LBA on visual development in deprived amblyopia rats might be related with up-regulating the expression of Bcl-2 and NGF , which could improve nutrition and growth of the nerve cells .

  11. 目的在视觉发育可塑性关键期内,探讨双眼形觉剥夺对大鼠视皮层神经元N-甲基-D-天冬氨酸受体亚单位1(N-methyl-D-aspartateReceptor1,NR1)的影响。

    Objective To investigate the effects of binocular form-deprivation on the expression of N-methyl-D-aspartate receptor subunit 1 ( NR1 ) of visual cortical neurons in rats during the critical period of visual plasticity .

  12. 视觉发育可塑性的电生理及分子生物学研究进展

    Advance in electrophysiological and molecular biology research on visual plasticity during development

  13. 弱视是小儿视觉发育敏感期常见的视觉紊乱。

    Amblyopia was the common ocular disorder in children visual development sensitive period .

  14. 视觉发育敏感期是弱视发生的危险期,也是逆转弱视的最佳时机。

    The sensitive period of visual development is a critical period for amblyopia .

  15. 视觉发育可塑性关键期终止机制的研究进展

    Advances in mechanism of ending the critical period of visual plasticity during development

  16. 结论儿童人工晶体眼仍存在视觉发育的近视化趋向。

    Conclusions The ocular growth of children after implantation of IOL still has a trend to myopia .

  17. 研究视觉发育敏感期及其可塑性,对弱视治疗具有重要的指导意义,并为弱视的防治提供了理论依据。

    Studies on the sensitive period and plasticity of visual development are important in the treatment of amblyopia .

  18. 结论采用胞二磷胆碱治疗超过视觉发育敏感期的大龄弱视安全有效。

    Conclusion CDP choline improved visual acuity in amblyopic patients beyond the sensitive period of the visual system .

  19. 视觉发育是脑研究的一个分支,也是眼科临床基础研究的重要方向。

    Visual development , a branch of neuroscience , is an important field for clinical and theoretical research in ophthalmology .

  20. 在儿童保健及小儿眼科工作中,定量评价婴儿的视觉发育非常重要。

    It ′ s important to detect visual acuity of infants quantitively in children ′ s health care and pediatric ophthalmology .

  21. 在视觉发育可塑性关键期内,异常的视觉环境(如暗饲养等)影响视皮层的可塑性。但是,哪些神经递质及受体参与了视皮层可塑性的改变仍需要进一步研究。

    During the critical period of visual development , abnormal visual environment such as dark-rearing influences the experience-dependent plasticity of visual cortex .

  22. 我们的结论是:(1)任何早发性眼科疾病,假如阻碍了正常的双眼视觉发育,则造成不对称的运动觉;

    We concluded that ( 1 ) motion asymmetry correlates closely with early-onset eye disorders that hinder the normal development of binocular vision ;

  23. 生后视觉发育存在“敏感期”,在此期间视觉神经系统具有经验依赖的可塑性。

    During a brief period in postnatal life , the existence of critical periods for experience-dependent plasticity has been clearly demonstrated for the visual system .

  24. 尽早去除形觉剥夺,保持或恢复视觉发育敏感期的正常视觉环境,有利于预防近视眼的发生。

    To eliminate form deprivation earlier is beneficial to maintain and restore a normal visual environment during the sensitive period of visual development in order to prevent myopia .

  25. 目的探讨双眼形觉剥夺对大鼠视觉发育可塑性关键期内视皮层神经元γ氨基丁酸电流的影响。

    Objective To investigate the effects of binocular form deprivation on the properties of inhibitory synaptic transmission of rat visual cortical neurons during the critical period of visual development .

  26. 结论随着视觉发育,正常大鼠视皮层神经元的γ氨基丁酸受体介导的抑制性突触传递增强,双眼形觉剥夺抑制这一变化,恢复视觉输入后可以逆转。(中华眼科杂志,2005413740)

    Conclusion With visual development , the synaptic transmission of visual cortical neurons in normal rats is strengthened , but it can be inhibited by binocular form deprivation and recovered by visual input .

  27. 目的:弱视是在视觉发育可塑性关键期内由于形觉剥夺和/或双眼视觉异常相互作用造成的单眼或双眼视功能发育障碍。

    Objective : Amblyopia is the monocular and binocular developmental disorder which due to the deprivation of form sense and / or the binocular abnormal interaction in the critically period of visual development .

  28. 弱视是在视觉发育可塑性关键期间,由于视觉细胞接受有效刺激不足,造成矫正视力低于正常,而眼科检查未能发现明显器质性病变。

    Amblyopia is a disorder referring to impaired vision without obvious pathological changes in ophthalmic examinations , which is resulted from insufficient visual information received by visual neurons during the critical period of rat visual development .

  29. 推测其治疗机理与中药可以增加眼局部血液供应,改善视网膜供养,促进黄斑区再发育、提高视敏度、延长视觉发育的敏感期有关。

    It was extrapolated that its treatment mechanism correlated with the effect of Chinese medicine , which can increase the ocular blood supply , improve support to retina and promote further development of macular and improve visual acuity and delay the sensitive period of visual development .

  30. 目的研究大鼠视觉发育可塑性关键期视皮层Ⅱ-Ⅲ层神经元的电学特性变化及其与大鼠生理年龄及视觉经验的关系,探索视皮层经验依赖性可塑性的突触和细胞机制。

    Objective To study the electrophysiological properties of postsynaptic neurons from layer ⅱ - ⅲ in normal rat visual cortex during the critical period of visual plasticity and its relationship with age and visual experiences and to explore the synaptic and cellular mechanisms of experience-dependent plasticity in visual cortex .