高分子右旋糖酐
- 网络High molecular dextran;HMWD
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经大鼠尾静脉注射10%高分子右旋糖酐10ml/kg,持续20天,复制成大鼠慢性微循环障碍模型,对照组尾静脉注射生理盐水。
Rats were injected with 10 % HMWD dextran 10 ml / kg via the tail vein every other day for 20 days to duplicate the model of chronic disturbance of rat 's microcirculation .
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结果高分子右旋糖酐造成了大鼠红细胞变形性降低(P0.05),聚集性增高(P0.01),膜流动性降低(P0.05)。
Result HMWD injection decreased red blood cells deformability ( P0.05 ) and liquidity of membrane ( P0.01 ) and increased erythrocytes aggregation ( P005 ) .
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颈静脉注射高分子右旋糖酐(dextran500)制备Wistar大鼠实验性DIC模型,通过淋巴学方法,观察DIC时大鼠淋巴循环的变化。
Experimental DIC model was replicated by injecting dextran 500 via jugular vein in Wistar rats . The changes in lymph circulation were observed in these rats with lymphatology methods .
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3min组:以同样浓度和剂量的高分子右旋糖酐颈静脉注射,3min注完。
Rats in the 3-minute group were given of intrajugular injection of high molecular dextran with the same dose and concentration within 3 minutes .
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Ⅲ组,在AEP模型的基础上静注10%高分子右旋糖酐(分子量110000)500mg/kg诱发AHNP。
Group III , AHNP was produced on the basis of the AEP model by adding an intravenous injection of a large dose of Dextran ( 110 , 500mg / kg ) .
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当归对高分子右旋糖酐诱导人红细胞聚集性增强的影响
Inhibition Effects of Angelica on Induced Human Erythrocytes Aggregation By Dextran
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模型复制剂:10%高分子右旋糖酐。
Duplicated model agent ; 10 % macromolecular dextran .
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高分子右旋糖酐所致大鼠弥散性血管内凝血的血液动力学变化
Hemodynamic changes in rats with disseminated inravascular coagulation induced by high molecular dextran
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用高分子右旋糖酐复制家兔高黏血症模型。
A hyperviscosity model was established by venous injection of high molecular weight dextran .
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用高分子右旋糖酐(MW260000)静脉注射,复制兔的急性微循环障碍模型。
Acute microcirculation disturbance was brought about in New Zealand rabbits by rapid injection of high molecular dextran ( MW . 260,000 ) .
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方法从耳缘静脉注射高分子右旋糖酐(dextran500)复制血瘀证家兔模型,观察其血液流变性和球结膜微循环改变以及丹龙醒脑片对该模型的影响。
Methods The blood stagnant syndrome model was reproduced by injecting dextran 500 into the marginal ear vein . The BCM and the hemorrheology properties were detected and compared .
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经静脉注射高分子右旋糖酐,复制循环微血栓及心电图表现异常的大鼠模型,观察肝素对微血栓的治疗作用。
A microthrombus and abnormal ECG rat model was reproduced through the intravenous injection of macromolecular dextran . The treatment of heparin for microthrombus in the circulation had been studied .
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结论:高分子右旋糖酐所致大鼠实验性弥散性血管内凝血具有明显的血液动力学异常,该模型应用范围广,效果稳定,其效果与注射速度无关。
Conclusion : there is obvious hemodynamic abnormity in rats with DIC induced by high molecular dextran , and the model can be widely applied with stable effect , which is not related with the injection speed .
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目的:应用高分子右旋糖酐复制大鼠弥散性血管内凝血模型,观察高分子右旋糖酐对大鼠血液动力学的影响,并了解其作用是否与注射速度相关。
AIM : Rat models of disseminated intravascular coagulation ( DIC ) were established with high molecular dextran to explore its effects on hemodynamic changes in rats and acknowledge whether the mechanism is related with the injection speed .
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活血化瘀试验:健康家兔10只,耳静脉注射10%高分子右旋糖酐,制成血瘀模型,先测定全血黏度,以大剂量黄桂灌肠剂灌肠后,再测全血黏度,观察血黏度的变化。
The death rate of the rats was observed . ( 3 ) Test of promoting blood circulation and relieving blood stasis : the model of blood stasis in rabbits was made by injecting 10 % high molecular dextran into the veins of the ear of 10 healthy rabbits .
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右旋糖酐蔗糖酶(EC.2.4.1.5)能以蔗糖为底物,将D-葡萄糖基转移到正在增长的葡聚糖链上形成高分子的葡萄糖聚合物右旋糖酐。
Dextransucrase ( EC . 2.4.1.5 ) synthesize high molecular weight glucose polymer dextran by transferring the D-glucosyl moieties to a growing glucan chain using sucrose as substrate .