迟发性神经毒性

目的探讨三邻甲苯基磷酸酯（TOCP）中毒引起迟发性神经毒性的机制。

AIM To explore the mechanism of tri-o-cresyl phosphate ( TOCP ) induced delayed neurotoxicity .

目的：采用甲胺磷作为有机磷化合物诱导迟发性神经毒性（OPIDN）的受试物诱发母鸡OPIDN。

Objective : To develop an organophosphorus ester induced delayed neurotoxicity ( OPIDN ) hen model .

甲胺磷诱发母鸡迟发性神经毒性模型

Delayed Neurotoxicity is not Induced by Methamidophos in Adult Hen

生物标志物用于有机磷农药的迟发性神经毒性研究

Acute Delayed Neurotoxicity Study of Organophosphorous Pesticides by Biomarker Methods

三甲苯磷酸酯的迟发性神经毒性评价及其对超微结构的影响

Evaluation of Delayed Neurotoxicity of Tricresyl Phosphate and its Effect on the Ultrastructure of Sciatic Nerve and Spinal Cord

此类化合物种类繁多，结构各异。那么，哪些可能产生迟发性神经毒性？

There are different types and variations in chemical structure of these compounds which are relative to delayed neurotoxicity .

结果表明，本实验未能通过经口及经皮两种染毒途径在母鸡上建立甲胺磷诱发的迟发性神经毒性模型。

The results indicate that delayed neurotoxicity is not induced by methamidophos administered orally and subcutaneously in adult hens .

三邻甲苯磷酸酯诱发迟发性神经毒性动物模型的机制初探颞叶癫癎实验模型中急性和迟发性神经元变性的特征及机制研究

Primary study on machanism of OPIDN induced by TOCP in hens The Characteristic Pattern and Related Mechanisms of Acute and Delayed Neuronal Degeneration in Experimental Models of Temporal Lobe Epilepsy