败血症性休克

心源性和败血症性休克的微循环变化

Microcirculatory alterations in cardiogenic and septic shock

发烧、寒战和局部感染症状提示败血症性休克，特别是免疫代偿病人。

Fever , chills , and focal signs of infection suggest septic shock , particularly in immunocompromised patients .

由于细菌毒素的作用，尤其是内毒素，败血症性休克的促炎症性反应强于其他休克。

Septic shock may be more proinflammatory than other forms because of the actions of bacterial toxins , especially endotoxin .

败血症性休克常发生在细菌感染后，以机体免疫系统释放出大量的致炎（炎症前）细胞因子为特征。

Septic shock often follows a bacterial infection , and is characterized by the overwhelming release of pro-inflammatory cytokines by the body 's immune system .

重度骨髓抑制易并发败血症、感染性休克等严重时可导致死亡。

Severe bone marrow suppression may predispose to sepsis , septic shock can be fatal in severe .