腹腔感染

大黄素对腹腔感染大鼠血浆白蛋白及TNF-α、IL-6影响的实验研究

Effect of Emodin on Serum Albumin and TNF - α、 IL-6 of Abdominal infection Rats

腹腔感染所致多器官损害与Toll样受体2基因表达的关系

The relationship between gene expression of Toll-like receptor 2 and multiple organ dysfunction in rats after abdominal infection

口服高渗氯化钠维生素C液对腹腔感染大鼠肠屏障的保护作用

Influences of oral hypertonic saline with vitamin C on gut barrier function of rats with abdominal infection

腹腔感染后抗氧化能力对血浆维生素A、E、C水平的影响

The influence of antioxidation on plasma vitamin A , E , C following intraabdominal sepsis in rats

全肠外营养加用维生素A、E、C对严重腹腔感染大鼠肠屏障功能的影响

Influence of vitamin A 、 E 、 C-added TPN on barrier of small bowel mucosa in severely intraabdominal septic rats

腹腔感染伴多器官功能障碍综合征时抗TNF抗体代谢调理作用的实验研究

An experimental study of metabolic intervention of anti-TNF antibody in intra-abdominal infection complicated by multiple organ dysfunction syndrome

结论TPN添加维生素A、E、C能改善腹腔感染后机体的抗氧化能力，减轻脂质过氧化损伤。

Conclusion Vitamins A , E , C with TPN may effectively ameliorate the oxidative injuries in peritoneal sepsis .

抗TNF抗体防治腹腔感染诱发多器官功能障碍综合征的作用

The role of anti-tumor necrosis factor antibody in the treatment of multiple organ dysfunction syndrome induced by intra-abdominal infection

目的观察完全胃肠外营养（TPN）添加维生素A、E、C对腹腔感染后抗氧化能力的影响。

Objective To study the effects of vitamins A , E , and C with TPN on antioxidation in peritoneal sepsis .

氟嗪酸（Ofloxacin）治疗复杂性尿路感染和腹膜透析腹腔感染

Ofloxacin Treatment of the Complicated Urinary Tract Infections and CAPD Peritonitis

病理状态下大鼠腹腔感染前后TNF-α和IL-6基因表达的实验研究

Experimental Study on the TNF - α and IL-6 Gene Expression in Pathological Rat Before and After Celiac Infection

严重腹腔感染大鼠组织Toll样受体2/4基因表达及其调节机制

Changes in Toll-like receptor 2 and 4 gene expression in vital organs in septic rats and their regulation mechanisms

结论：TPN加用维生素A、E、C对严重腹腔感染大鼠肠屏障功能有一定的保护作用。

Conclusions : These results suggest that vitamin A 、 E 、 C added TPN can effectively protect the barrier of gut mucosa in infected rats .

结果：SAP早期手术和延期手术的腹腔感染发生率分别为76.71%和21.82%（P<0.005）；

Results : The infection rates of early operations and postponed operations are 76.71 % and 21.82 % respectively ( P < 0.005 ) .

结论联合使用环氧化酶抑制剂Asp和β-内啡肽受体拮抗剂Nal可以调理腹腔感染大鼠TPN时的蛋白质代谢，减少蛋白质丢失。

Conclusion Asp combined with Nal may intervene protein metabolism in TPN and reduce protein loss in sepsis .

免疫抑制大鼠腹腔感染致MODS发生机制及氨茶碱治疗的实验研究

Experimental Study on the Mechanisms and the Therapeutic Effect of Aminophylline for MODS Induced by Peritonitis in Rats with Immunosuppression

本研究旨在探讨重组人生长激素（rhGH）对腹腔感染引起的低白蛋白血症的治疗作用。

The present study was aimed to explore the effect of recombinant human growth hormone ( rh GH ) on hypoalbuminemia in peritoneal sepsis .

方法采用大鼠盲肠结扎并穿孔（CLP）造成腹腔感染。

Methods The cecal ligation and perforation ( CLP ) was utilized to make the abdominal infection in rats .

ERCP操作使胆汁内细菌阳性检出率增高，并可能是胰头癌根治术后腹腔感染的潜在危险因素。

ERCP caused positive bile culture and may be potential risk factor of intra-abdominal infection after radical resection of pancreatic adenocarcinoma . 3 .

结论：大黄素能减轻CLP所致腹腔感染大鼠的炎症反应和肠粘膜的损伤。

Conclusion Emodin can reduce the inflammatory response and extenuate the damage to intestinal mucosa in abdominal infection of rats .

结论：1.不同年龄段腹腔感染大鼠脾切除前后TNF-α、IL-10的变化水平基本一致。

For celiac infection rats of different ages , the changes in their TNF-a and IL-10 level before and after splenectomy are very similar . 2 .

脾切除导致腹腔感染晚期大鼠体内TNF-α降低及IL-10的升高，使其更易发生免疫抑制。

The reduction of TNF-a and increase of IL-10 level in celiac infection rats of late stage are more likely to induce immunosuppression after splenectomy . 3 .

结论一定剂量的KET（50mg/kg）可显著降低腹腔感染脓毒症大鼠死亡率。

Conclusions Ketamine at a given dose can decrease significantly the mortality of septic rats .

目的探讨Toll样受体2（TLR2）基因表达与严重腹腔感染所致多器官损害的关系。

Objective To investigate the relationship between gene expression of Toll-like receptor 2 ( TLR2 ) and multiple organs dysfunction after severe abdominal infection .

应用盲肠结扎穿孔法（CLP）制备腹腔感染大鼠模型，同时给予外源性生长激素；

The intra_abdominal infection model was made by cecal ligation and puncture ( CLP ) and the infected rats were treated with exogenous growth hormone .

腹腔感染致呼吸窘迫综合征的动物模型Gq蛋白及RGS在急性呼吸窘迫综合征中的作用机制研究

Respiratory distress syndrome due to intraperitonealinfection : Study in animal models The Mechanism of Gq Protein and RGS on ARDS

所有患者由ICU医生行重要器官功能支持治疗；对有明确腹腔感染病灶，经非手术治疗效果不理想或病情加重的患者进行外科干预。

All patients by doctor of ICU support for life and organ function ; the lesions of abdominal infection , surgical intervention by non operation treatment effect is not ideal or sicker patients .

目的了解广州地区腹腔感染产ESBLs大肠埃希菌和肺炎克雷伯菌的耐药性及ESBLs基因型分布。

Objective To investigate the drug resistance of Escherichia coli and Klebsiella pneumoniae producing extended-spectrum β - lactamases ( ESBLs ) in intraabdominal infections and analyze the genotypes of ESBLs .

我们应用腹腔感染诱发多器官衰竭（MOF）的动物模型观察不同热量全肠外营养（TPN）对MOF兔的器官功能、氮平衡和蛋白质分解代谢的影响。

Rabbits with MOF induced by intraab-dominal sepsis were used to observe the effect of TPN with different amount of calorie on organ function , nitrogen balance and protein catabolism .

目的：观察抗TNF抗体对腹腔感染（IAI）伴多器官功能障碍综合征（MODS）时的代谢调理作用。

Objectives : To observe the effect of metabolic intervention of anti TNF antibody on the hypermetabolism occurred in intra abdominal infection ( IAI ) complicated by multiple organ dysfunction syndrome ( MODS ) .