淤血

以实质器官淤血、出血、变性和胃肠炎为主要病理变化。

The major pathological changes were extravasated blood in parenchymal .

2号犬存活1小时，左肺静脉及左心房形成血栓，出现肺淤血、肺水肿导致死亡；

The 2nd dog survived for 1h and thrombus formed in left pulmonary vein and left atrium cause extravasated blood and pulmonary edema , which leaded to the death ;

活体肝移植术后供、受者肝静脉淤血及肝再生的多层螺旋CT研究

Hepatic Venous Congestion and Liver Regeneration of Donor-recipients after Living Donor Liver Transplantation : Multi-slice Spiral CT Study

盆腔静脉淤血综合征和健康育龄女性盆腔静脉的MRI研究

MRI Study of Pelvic Veins in Patients with Pelvic Congestion Syndrome and Healthy Women in Their Reproductive Years

淤血性肝硬变的MSCT、MRI诊断价值

Diagnostic Value of MSCT and MRI for Stasis Cirrhosis

活体肝移植术后肝淤血的多层螺旋CT评价及原因分析

Hepatic Venous Congestion after Living Donor Liver Transplantation ( LDLT ) with Right Lobe Graft : Postoperative Evaluation and Reason Analysis Using Multi-detector Row CT

光镜观察：C组和D组脊髓损伤大鼠的组织学改变主要有①肝中央静脉淤血，肝细胞浊肿；

Histological findings : The histological damages in rats with spinal cord injury in group C or in group D mainly included : ( 1 ) congesting in central vein , swelling of liver cells ;

B组随着移植后时间的延长，肝组织病变逐渐加重，出现不同程度的肝淤血、肝窦结构破坏、肝细胞变性及坏死；

In group B , along with the time going on , the histopathologic changes gradually aggravated , the changes included different degrees hepatic congestion , configuration destroy of hepatic sinusoid , degeneration and necrosis of liver cell ;

深静脉血栓（Deepvenousthrombosis，DVT）是指血液在静脉腔内不正常的凝结，导致血液回流障碍而引起组织淤血。

The deep venous thrombosis ( DVT ) is the disease that venous blood has curdled abnormally , causing the blood can ' flow back and tissues have gores .

肉鸡腹水综合征（AscitesSyndrome，AS）是一种非传染性疾病，临床上以显著的腹腔积水、右心室肥大／衰竭、肺充血、水肿以及肝淤血为主要特征。

Ascites Syndrome ( AS ) is a non-communicable diseases . Clinically characterized by significant ascites , right ventricular hypertrophy / failure , pulmonary congestion , edema and liver congestion / swelling .

IUGR组的胎盘与对照组胎盘相比，绒毛稀少，绒毛内血管减少，血管淤血、出血的比例明显高于对照组（P0.05）。

The percentage of gore and hemorrhage in placenta were higher in the IUGR group than that of the control group ( P0.05 ) . 4 .

结果：RSV肺炎病理改变严重，肺泡毛细血管出现淤血、微血栓。

Results : Pathological changes in mice with RSV pneumonia were serious with extravasation of blood and embolism in alveolar capillary .

对照组6h肺泡间质淤血、水肿，嗜中性粒细胞聚集和浸润；

In the control groups at hour 6 there were congestion and hydroncus in alveoli pulmonis mesenchyma , aggregation and infiltration of neutrophil .

PGE1干预可明显减轻淤血再灌注对肠粘膜结构损伤和保护肠粘膜对葡萄糖的吸收能力。

When the rats treated with Lipo-PGE1 the intestinal injury was remarkably attenuated and the ability to absorb glucose was greatly enhanced .

目的：研究前列地尔脂微球载体注射液（Lipo-PGE1）对大鼠小肠淤血性损伤的保护作用。

Objective : To study protective effect of Lipo-PGE_1 for injection on intestinal congestion in rats .

结论：肠道急性淤血再灌注后肠道微循环的开放数明显减少，而Lipo-PGE1的干预可以明显改善微循环，减轻肠道组织的损伤。

Lipo-PGE1 could protect effectively microcirculation and the injury caused by acute intestinal venous congestion reperfusion .

B组肺体积增大，表面淤血，病理改变以肺间质病变为主，肺组织毛细血管明显扩张、充血，肺泡隔增宽，大量白细胞渗出、聚集。

In group B , lung increased and surface congested . The pathological changes were as following : interstitial lung disease , the dilation and congestion of pulmonary capillaries , widened alveolar septum , the leakage and aggregation of a large number of white blood cells .

【结论】①肠静脉回流阻断60min引起的肠道淤血可导致肠粘膜屏障损伤。

[ Conclusions ] ① The intestinal congestion followed by vein clamping can cause gut barrier damage after 60 min.

凯时（Lipo-PGE1）对大鼠小肠淤血再灌注损伤的影响

Effect of Lipo-PGE_1 on Intestinal Injury Due to Blood Reperfusion after Venous Congestion in Rats

于小肠淤血前、淤血60min和再灌注60min时分别取末段回肠标本，观察和比较各时间点小肠黏膜的损伤情况，同时测定肠组织中MDA含量利MPO活性的变化。

Ileum tissue was harvested respectively for histological examination and MDA and MPO determination at each time of before congestion , congestion for 60 minutes and reperfusion for 60 minutes .

目的：通过阻断后再开放门静脉建立大鼠小肠的淤血再灌注模型，研究Lipo-PGE1对淤血再灌注损伤肠道结构和功能有无保护作用。

Objective : To study the effect of Lipo-PGE1 on intestinal injury due to blood reperfusion after venous congestion by portal occlusion in rats .

目的研究肠淤血大鼠肠神经系统内乙酰胆碱酯酶（AchE）神经元和一氧化氮合酶（NOS）神经元的改变，以探讨肠淤血影响肠道运动功能的神经机制。

Objective To explore the nervous mechanism of intestinal function disorder after intestine congestion through observing the changes of acetylcholinesterase ( AchE ) neuron and nitric oxide synthase neuron ( NOS ) expression in nervous system of rat intestinal tract .

方法SD大鼠20只，雌雄各半，采用阻断门静脉60min开放60min的方法建立大鼠小肠淤血再灌注模型；

Methods The model of congestion-reperfusion of intestine was established by means of the occlusion of portal vein for 60 minutes and then reflow for 60 minutes in 20 Sprague-Dawley rats .

d30及d45病理组织学检查发现每组均有部分动物出现肝、肾淤血，轻度细胞水肿，可能系动物的隐性感染而非药物所致。

The histopathologic examination showed congestion and slight edema of liver and kidney in each group .

经病理检查发现：①小鼠肺泡间隔中毛细血管腔内广泛存在着HAP的聚合体，并有广泛的肺泡间隔内淤血及肺泡水肿；

The pathological examination indicated that : ① There was large quantities of embolism of HAP combined corps obstructed in the capillary vessels of lung , and the alveolar interval congestion and alveolar edema were found extensively ;

且NS－1亚型和NS－2型均可出现梗阻型肾图，而NS－2型出现梗阻的机会较多，多表现为阳虚，且淤血程度重.治疗难度大。

Both NS - I and NS - I may appear on the obstructive renogram in NS - I are more than that in NS - I , The former often manifests deficiency-Yang and blood stasis and its treatment is extremely difficulty .

在60min内肠淤血性的损伤不会引起肠腔内内毒素的大量移位及TNF-α的释放。

The degree of gut barrier damage caused by intestinal congestion within 60 min is not severe enough to cause endotoxin translocation and tumour necrosis factor alpha released .

脂质过氧化反应参与了大鼠小肠组织的淤血再灌注损伤过程，但尚不能确定Lipo-PGE1减轻淤血再灌注损伤的机理与此有关。

Hyperoxidation of lipid in intestine happened during the congestion and reperfusion . But there was no reason by which Lipo-PGE1 could alleviate the hyperoxidation of lipid in intestine .

CR组和Lipo-PGE1治疗组均制作肠道急性淤血再灌注模型。模型制作：先阻断门静脉45min，再恢复血流60min。

To induce intestinal venous congestion reperfusion model , portal vein was clamped for 45 min , then unclamped for 60 min. This reperfusion model was made in CR group and in Lipo-PGE1 group .

H.E.染色结果显示，病理组织学变化主要表现为肾有淤血，肾小管上皮细胞发生变性和坏死。

Gizzard and proventriculus were hemorrhage . H.E.staining results showed that the most pathological changes were congestion in kidney , denaturation and necrosis of epithelial cell of renal tubule .