基底核

3讨论CT上表现为双侧基底核对称性低密度病变的疾病较多，主要包括代谢性疾病、感染性疾病和中毒性疾病。

Conclusion On CT , many diseases are manifested as symmetrical low density lesions at bilateral basal nuclei .

血氨均正常；头颅CT均显示显著脑肿胀，可见对称性基底核低密度病变。

Blood ammonemia was normal , brain CT scans revealed peripheral or basal nuclei low-density areas .

CT上表现为基底核区局限性点状钙化。

The CT scan showed localized punctuate calcification in basal ganglia .

MRI脑图像基底核区部位的分割算法研究

The Study of Basal Ganglia Segmentation Algorithms Based on Brain MRI

PET显示模型侧基底核区低代谢，与术前相比明显不同。

PET showed the low metabolism in the basal ganglia region of the injected side .

目的分析CT上表现为双侧基底核对称性低密度的各种疾病，以加深对该CT表现的认识。

Objective To make a further understanding of CT manifestations of various diseases at bilateral basal nuclei displaying symmetrical low density lesions .

结论：中枢组胺可以明显改善前脑基底核破坏所致大鼠记忆障碍，其作用主要与H1受体与胆碱能神经相关。

CONCLUSION : Histaminergic neurons play an important role in learning and memory via H1-receptor , and its action may be due to cholinergic neurons .

家兔杏仁基底核影响V1区神经电活动的研究

Basal Amygdaloid Influence on Electrical Activities of Visual Cortex in Rabbits

大鼠扣带回&丘脑腹侧基底核投射的HRP研究

HRP Labeling Study on Anterior Cingulate Cortex & Ventrobasal Nucleus of the Thalamus Projection of the Rat

手术后CT检查发现基底核区梗死3例，手术造成的皮质损伤3例，基底核区出血1例。

Postoperative CT scan demonstrated basal ganglion infarction in 3 cases , cortical injury caused by surgical manipulation in 3 cases and postoperative intra-parenchymal hematoma in the basal ganglion in one case ;

Meynert基底核的老龄改变及相关因素

Age-related Changes of Basal Nucleus of Meynert and Its Related Factors

方法：SD大鼠脑右侧基底核部注入淀粉样β蛋白（Aβ）和鹅膏氨酸。艹覃混合物以造成拟似痴呆模型。

METHODS : A mixture of β amyloid ( A β) and ibotenic acid ( IBO ) was injected directly into right basal brain ganglia of SD rats to build the simulant dementia model .

Meynert基底核注射冈田酸致大白鼠空间记忆障碍

Injection of Okadaic Acid into the Nuclei Basalis of Meynert Induced Spatial Memory Deficit in the Rat

不同年龄组抑郁症模型鼠Meynert基底核病理形态学研究

Investigation on Pathomorphology of Meynert Basal Nuclei Nerve Cell in Depression Models of Different Cotemporary Sd Rats

用HRP逆行示踪法证实了大鼠扣带回前部丘脑腹侧基底核投射通路。

Using HRP labeling techniques , we have demonstrated that there was a direct projection from anterior cingulate cortex to ventrobasal nucleus of the thalamus in the rat brain .

结果表明：留针和电针刺激对对侧大脑半球皮质、对侧丘脑、同侧基底核和双侧小脑的作用有显著性差异（P＜0.01），电针者血流功能变化率高于留针者。

Results showed a significant difference in effect of needle retention and electro-acupuncture stimulation on contralateral cerebral hemisphere cortex and thalamus , ipsilateral basal ganglion and bilateral cerebella , the change on cerebral blood flow induced by electroacupuncture was greater than that induced by the former method .

目的探讨一次性双侧基底核大细胞（nbM）区域β-淀粉样多肽（AβP）25-35注射对大鼠空间学习行为的影响。

Objective To investigate the effects of bilaterally injection of amyloid-beta ( 25-35 ) peptide ( A β P25-35 ) to the nbM areas on the spatial learning and memory in rats .

D-半乳糖合并Meynert基底核损毁对海马长时程增强和突触形态的影响

Effects of D-galactose combined with lesions of nucleus basalis of Meynert on hippocampal long-term potentiation and synaptic morphology

目的探讨儿童基底核钙化（BGC）的相关疾病、主要病因和临床特点。

Objective To explore the related diseases , main causes and clinical features of children with basal ganglia calcification ( BGC ) .

同侧皮质及基底核区GFAP染色阳性细胞逐渐增多（P<0.001），细胞体积增大，突起增多。

The GFAP positive cells gradually increased in the same lateral cortex and basal ganglia ( P < 0.001 ) . The cell size enlarged , and the number of dendrites increased .

方法采用♂Wistar大鼠右侧基底核注入50μl自体全血的方法建立模型，并在模型建立后6,24,48或72h处死大鼠。

Methods Male Wistar rats received an injection of 50 μ lautologous whole blood into the right basal ganglia and were killed 6 , 24 , 48 or 72 h later .

结果：HRP注射到听皮层，同侧杏仁外侧核、杏仁基底核、杏仁前区和杏仁前皮层观察到逆行标记细胞；

Results : After HRP injection into auditory cortex , some retrogradely labelled cells were observed in lateral amygdaloid nucleus ( LA ), basal amygdaloid nucleus ( BA ), anterior amygdaloid area , and anterior cortical amygdaloid nucleus .

目的：应用喹啉酸损毁Meynert基底核建立非遗传性阿尔茨海默病大鼠模型。

AIM : To establish rat models of non-hereditary Alzheimer 's disease ( AD ) by dama ging bilateral nucleus basalis of Meynert ( NBM ) with quinolinic acid .

当HRP注入丘脑腹侧基底核时，在同侧扣带回前部观察到标记的细胞，标记细胞密度背侧区比腹侧区高。

When HRP was injected to ventrobasal nucleus of the thalamus , labeled cell bodies were observed in anterior cingulate cortex . The density of labeled cell bodies was higher in the dorsal area than that of in the ventral area .

目的探讨IL-1β对大鼠胆碱能神经元的损伤作用及机制。方法将IL-1β缓慢释放颗粒（IL-1βpellet）植入大鼠右侧Meynert核（nbM，基底核），复制胆碱能系统受损的动物模型；

Objective To observe the effect and mechanism of IL 1 β on cholinergic neurons in the rat . Methods IL 1 β pellet was put into right side of nucleus basalis of Meynert to make cholinergic system damaged .

Ch4均匀分布于Meynert基底核（NB），占68.1%，胞体深染，形态多样，直径14～17μm，突起少。

Ch4 distributed evenly in Meynert nucleus basalis ( NB ), proportioned 68.1 % . The cells were stained heavily , 14-17 # μ m in diameter , with multiplicate shapes , and the cellular processes were sparse .

缺血30min，15只成功模型的PET结果即显示双侧基底核、颞叶、顶叶皮质葡萄糖代谢率的比值（右侧/左侧）下降，且随缺血时间延长下降越明显；

After 30 minutes of ischemia , the PET results of the 15 successful models showed that the ratio of glucose metabolic rate decreased in bilateral basal ganglias , temporal and occipital cortexes , and decreased significantly with the ischemic time prolonged .

结果发现，Meynert基底核注射OA后，Ach水平降低，τ蛋白在Ser198/Ser199/Ser202，Ser396/Ser404位点发生过度磷酸化，并伴有大鼠空间记忆障碍。

It was found that injection of okadaic acid into the Meynert nucleus basalis of rat brain induced hyperphosphorylation of tau at Ser-198 / Ser-199 / Ser-202 and Ser-396 / Ser-404 epitope , and decreased acetylcholine level accompanied with special memory deficit .

用Photoshop软件，对大脑灰质和白质、基底核团、脑室及海马等结构进行了半自动分割，并在PC机上用表面绘制和体绘制方法进行了三维重建。

Semi-automated segmentation and Photoshop software were selected to segment cerebral cortex , white mater , basal nuclei , lateral ventricle and hippocampus et al . On personal computer , the segmented structures were reconstructed in three-dimensions with volume rendering reconstruction and surface rendering reconstruction .

研究背景：早在1904年，Schmorl对1例因重症黄疸而死亡的新生儿进行尸解发现脑基底核被黄染，并首次命名为核黄疸（kernicterus）。

Background In 1904 , Schmorl held an autopsy on the remains of one neonate died of severe jaundice and found that the brain basic nuclear was yellow-dyed and named this disease as kernicterus .