hyperpolarization

The high K + solution could inhibit the Ach-induced hyperpolarization .

高钾则可取消Ach的内皮依赖性超极化。

Experimental Study of Hyperpolarization of 11,12-EET on porcine coronary smooth muscle cell

11，12-环氧花生三烯酸对猪冠状动脉平滑肌细胞超极化作用的实验研究

Role of Smooth Muscle Cell Hyperpolarization in the Decreased Vascular Reactivity by Peroxynitrite in Rats

平滑肌细胞膜超极化在过氧亚硝酸阴离子降低大鼠血管反应性中的作用

Endothelium - Dependent Hyperpolarization to Acetylcholine in Rabbit Pulmonary Artery

乙酰胆碱致兔肺动脉内皮依赖性超极化作用

Effects of Genistein on Endothelium - dependent Hyperpolarization in Aorta of Ovariectomized Rats

三羟异黄酮对去势雌性大鼠主动脉内皮依赖性超极化的影响

Rapid Hyperpolarization and Apoptosis of Murine Macrophages Furthermore , hyperpolarization was found in some root cells .

小鼠巨噬细胞膜快速超极化和凋亡有小部分水稻根表现为超极化。

Reactive oxygen intermediates and mitochondrial hyperpolarization in lymphocytes from patients with liver involvement in systemic lupus erythematosus

狼疮性肝病患者外周血淋巴细胞活性氧和线粒体膜超极化的研究

Suppressed the inward rectifier K + current ( I KI ) significantly , especially at hyperpolarization ;

明显抑制内向整流钾电流（IK1），尤其在超极化时；

Role of large conductance calcium-activated potassium channel of arteriolar smooth muscle cells in cell membrane hyperpolarization after severe hemorrhagic shock

平滑肌大电导钙激活钾通道在重症失血性休克细胞膜超极化中的作用

Inhibition , local hyperpolarization and hyper-depolarization complex .

抑制性诱发反应为超极化局部电位和超极&除极复合电位。

The change of ultrastructure and concentration of ATP of isolated rabbit 's heart after hypothermic preservation by X-J hyperpolarization cardiac preservation solution

X-J心脏保存液对离体心脏不同保存时限ATP及超微结构的影响

The results suggest that the potassium Channels not the Na / K pump might contribute to the Ach-induced hyperpolarization in rabbit pulmonary artery .

提示：Ach引起的家兔肺动脉内皮依赖性超极化可能由细胞膜上钾通道开放所致，而和Na／K泵无关。

Objective : The electrophysiological mechanisms underlying hypoxic hyperpolarization of hippocampal neurons were studied to reveal the role of hypoxic hyperpolarization in brain injuries .

目的：研究缺氧超极化在缺氧海马神经元中的电生理机制，以揭示缺氧超极化在脑损伤中的作用。

In addition to the membrane potential , the [ K + ] o was another channel - gating factor and its effect was more significant especially under greater membrane hyperpolarization .

除膜电位外，〔K＋〕o是此型通道的另一个门控因素，且在膜电位超极化增高的情况下，其作用更加显著。

Results The endothelium-dependent hyperpolarization decreased significantly in group B when compared with group A , C and D ( P < 0.05 ) and improved after giving estradiol and genistein .

结果去势大鼠主动脉内皮依赖性超极化较假手术组减弱（P<0.05），补充雌二醇和三羟异黄酮后，内皮依赖性超极化作用明显改善。

Cardiac pacemaking is controlled by an inward current called I f , which is activated by membrane hyperpolarization of pacemaker cells in the sinoatrial node .

心脏的起搏受控于一类存在于心脏窦房结被称作If的离子通道，该通道是被细胞膜的超极化所激活。

Ionically , Inhalational anesthetics could act on some sites of subunits of K2P channels and then open the channels and generate hyperpolarization current .

吸入麻醉药作用于神经系统双孔钾通道部分亚型的某些位点，使该通道开放，而产生超极化电流。

The results showed all the three kinds of ligands caused macrophage membrane potential hyperpolarization , cytosolic acidification and lysosomal alkalization , but reached their stabilized conditions at different times .

结果显示三种配体均导致细胞膜电位超极化，胞浆pH降低，溶酶体pH升高，三个生理参量趋于稳定时间稍有不同。

Conclusions Myocardial energy preservation was better and recoveries of oxygen metabolism were rapid after hyperpolarization of the heart , and no difference was found between blood and crystalloid hyperpolarized arrest .

结论超极化停搏组的心肌能量保存良好，氧化代谢恢复迅速，而血液和晶体超极化停搏之间对心肌能量代谢无明显影响。

Besides cyclooxygenase and nitric oxide synthase , another distinct endothelial pathway , endothelium-dependent hyperpolarization factor ( EDHF ), is involved in relaxation of the vascular smooth muscle cells .

内皮依赖性超极化因子（endothelium-depen-denthyperpolarizingfactor，EDHF）是血管平滑肌内皮源性舒张反应中通过非环氧合酶、非一氧化氮合酶途径的舒张因子。

The results showed that HCG depolarised membrane potential of toad oocytes , while tyrosine directly caused membrane , hyperpolarization , and inhibited the depolarization induced by HCG .

结果表明，HCG对卵母细胞膜电位有去极化作用，酪氨酸可以使膜电位呈现超极化反应，并能抑制HCG的去极化作用。

In some cases when E_t exceeded 100 mV , both hyperpolarization and after-depolarization disappeared , and a " normal " action potential of VF was obtained .

有时在动作电位超过100mV或更高时，超极化及后去极化均消失，而皇正常心室肌动作电位图形。

Fluorescence measurements showed hyperpolarization could not induce the increase of [ Ca2 + ] i. On the contrary , [ Ca2 + ] I was decreased in some cells ( 13:3 ) . 4 .

荧光测钙显示超极化不能引起[Ca2+]i的升高，部分细胞表现出[Ca2+]i降低（13：3）。

This , rather gradual hyperpolarization has been attribute to the increasing of Na + / K + - pump activity , and K + - current or the decreasing of the Cl - reversal potential .

静息膜电位的超极化可能与Na+/K+泵活性增强以及K+电流的增加或Cl-翻转电位的降低有关。

Avermectin insecticides can cause the release of gamma-aminobutyric acid ( GABA ) and enhance the affinity of GABA with GABA receptors , leading to chloride influx and postsynaptic hyperpolarization .

阿维菌素类杀虫剂主要作用于γ氨基丁酸（GABA）受体，它能促进GABA的释放，增强GABA与GABA受体的结合，使氯离子内流增加，导致突触后膜超级化。

In calcium-free solutions , the reversal potential of the IK currents was shifted to the direction of hyperpolarization , which was very close to the equilibrium K + potential based on the Nernst equation .

在无钙外液中，Ik电流的反转电位向超极化方向明显移位，更接近于按照Nernst方程得出的K+理论平衡电位；

Results The INa current density-voltage (ⅰ - ⅴ) relationship curves of the M cells decreased significantly after simulated ischemia , The INa inactivation curves shifted in the hyperpolarization direction , there were no significant differences in the INa recovery curves from inactivation .

结果模拟缺血后，左心室M细胞的INa电流密度-电压（Ⅰ-Ⅴ）关系曲线呈时间依赖性下移，INa稳态失活曲线呈时间依赖性左移，失活后再恢复无明显变化；

We found that acetylsalicylic acid ( ASA ) can induce marked membrane potential hyperpolarization on dorsal root ganglion neurons ( DRG ) in rats by intracellular recording long before , which confirmed that ASA can produce analgesic effect of peripheral nerve end-brush .

以往用细胞内微电极技术研究发现乙酰水杨酸可引起大鼠背根神经节神经元膜产生明显的超极化反应，即能产生外周神经镇痛作用。

The cellular membrane potential hyperpolarization or depolarization is caused by some merterrials ( such as endothelium-derived hyperpolarizing factor , acetylcholine , bradykinin , intercellular second messager and so on ) which take part in the informational communication between VEC and VSMC .

内皮源性超极化因子、乙酰胆碱、缓激肽、第二信使等物质亦可引起内皮细胞或/和平滑肌细胞细胞膜的超极化或去极化，参与血管内皮细胞与平滑肌细胞间的信息传递。