ccl5

Result : this paper only reports differed CcL5 mRNA expressions .

结果：仅对筛选出差异表达基因谱中的CcL5mRNA进行报告。

CCL5 is known to activate factors that suppress HIV infection of human immune cells .

众所周知，CCL5可激活细胞因子抑制艾滋病毒感染人体免疫细胞。

Mesenchymal stem cells promote the migration and proliferation of Saos-2 cells through CCL5 / CCR5 axis

间充质干细胞通过CCL5/CCR5信号途径促进骨肉瘤Saos-2细胞迁移和生长

RESULTS : RA synoviocytes spontaneously produced a higher level of CCL5 than OA synoviocytes did ( P < 0.05 ) .

结果：RA滑膜细胞体外自发产生CCL5水平高于OA滑膜细胞（P<0.05）；

Tagging of Apoptotic Cells by the β - chemokine CCL5 / RANTES Stimulates Phagocytosis by Macrophages

β-趋化性细胞因子CCL5/RANTES促进巨噬细胞对凋亡细胞的吞噬

Objective : To study the relationship between the liver inflammation degree and the expression of CC chemokine ligand 5 ( CCL5 ) of the liver in patients with !

目的：探讨慢性乙型病毒性肝炎（慢乙肝）患者肝组织CC类趋化因子配体5（CCche-mokineligand5，CCL5）水平与肝脏炎症程度的关系。

With further investigation , they concluded that morphine induces production of the protein CCL5 , which they discovered is released by astrocytes , a type of brain cell .

进一步研究后，得出的结论是吗啡可诱导由星形胶质细胞（一种脑细胞）释放的CCL5蛋白的生成。

The relationship of serum CCL5 level with joint swollen index , erythrocyte sedimentation rate ( ESR ) and C-reactive protein ( CRP ) in rheumatoid arthritis patients was statistically analyzed with linear correlation regression analysis .

用直线相关回归分析法分析类风湿关节炎患者血清CCL5水平与关节肿胀指数、血沉、C反应蛋白之间的相关性。

AIM : To observe the source of production of CC chemokine CCL5 in vitro of rheumatoid arthritis patients and the relationship of level of CCL5 with symptom of rheumatoid arthritis and index of laboratory examination .

目的：观察趋化因子CCL5在类风湿关节炎患者体内的产生来源，以及CCL5水平与类风湿关节炎的症状和实验室检查指标的相关性。

RANTES ( regulated upon activation normal T cell expressed and secreted ; CCL5 ) is a member of the - CC - subfamily of chemokines , mainly generated by CD8 + T cells .

受激活调节正常T细胞表达和分泌因子（regulateduponactivationnormalTcellexpressedandsecreted，RANTES），是趋化性细胞因子CC亚族成员，主要由CD8~+T淋巴细胞分泌。

AIM : To determine the inhibitory effects of methotrexate ( MTX ) and tripterygium glycosides ( TG ) on chemokine CCL5 production in synoviocytes isolated from patients with rheumatoid arthritis ( RA ) .

目的：探讨甲氨喋呤（MTX）和雷公藤多甙（TG）对类风湿关节炎（RA）滑膜细胞产生CC趋化因子受体5配体（CCL5）的影响。