肌醇

增加B族维生素和肌醇含量也有利于胚状体的诱导。

Increasing content of B vitamins and inositol could also benefit to embryoid induction .

小鼠成纤维细胞中PLC水解肌醇磷脂的数学模型

A mathematical model of inositol phospholipid hydrolyzed by PLC in mouse fibroblasts

磷脂酰肌醇-3-激酶参与过度机械通气诱导的核因子-κB激活

Phosphatidylinositol-3-kinase participates in the activation of NF - κ B induced by hyperventilation

反义磷脂酰肌醇3-激酶寡脱氧核苷酸抑制白介素-1诱导的NF-κB活化

Antisense PI 3 kinase oligodeoxynucleotide blocks IL 1 stimulated NF κ B activation

外周血B细胞上糖肌醇磷脂连接蛋白的表达在阵发性睡眠性血红蛋白尿症诊断中的应用

Estimation of Expression of Glycosylphosphatidylinositol-anchored Protein in BLymphocytes for Diagnosis of Paroxysmal Nocturnal Hemoglobinuria

蛋白激酶B及其在磷脂酰肌醇3-激酶介导的信号转导中的作用

Protein Kinase B and Its Role in the Signal Transduction Pathway Mediated by Phosphoinositide 3-Kinase

自发性高血压大鼠G蛋白&肌醇磷脂途径的改变

Alteration of G Protein , Phosphoinositide Turnover in Spontaneously Hypertensive Rat

目的：构建小鼠成纤维细胞中磷脂酶C水解肌醇磷脂的数学模型。

AIM : To establish a mathematical model of inositol phospholipid hydrolyzed by phospholipase C in mouse fibroblasts .

它是组成，其中大部分B族维生素，磷酸，胆碱，亚油酸和肌醇。

It is composed mostly of B vitamins , phosphoric acid , choline , linoleic acid and inositol .

嵌合受体失去与G蛋白偶联的能力，也不能产生磷酸肌醇水解。

But the chimeric receptor failed to couple to G-protein and subsequent stimulation of PI turnover .

利用TAP标记分析酵母糖基磷脂酰肌醇转酰胺基酶复合物

Using the TAP tag strategy to analyze yeast glycosylphosphatidylinositol transamidase complex

肌醇脂质和cAMP在血小板活化因子诱导血小板聚集中的作用

Effect of inositol lipid and cAMP on PAF-induced platelet aggregation

人骨髓基质细胞中糖基化磷脂酰肌醇特异性磷脂酶DcDNA的克隆

Cloning of Glycosylphosphatidylinositol-Specific Phospholipase D cDNA from Human Bone Marrow Stromal Cells

竞争性RT-PCR法定量检测糖基化磷脂酰肌醇特异性磷脂酶D基因的表达

Competitive RT - PCR assay for quantification of GPI-PLD gene expression

促红细胞生成素经磷酸酰肌醇-3-激酶/蛋白激酶B途径增强自体骨髓间充质干细胞移植治疗大鼠心肌梗死的作用

Erythropoietin enhances the therapy potency of autologous bone marrow stromal cells in a rat heart infarction model via phosphatidylinositol-3-kinase / Akt pathway

磷脂酰肌醇特异性磷脂酶C对内毒素血症大鼠肝组织CD14表达的抑制作用

Inhibitory role of PI-PLC on CD14 expression in rat liver tissue during endotoxemia

酵母菌磷脂酰肌醇（PI）的生物合成及其重要的生理学功能

Biosynthesis of Phosphatidylinositol and its Physiological Functions in Yeasts

目的探讨甲状腺腺瘤疾病与磷脂酰肌醇3激酶/蛋白激酶B（PI3K/Akt）信号转导途径之间的关系。

Objective To explore the relationship between thyroid adenoma and phosphatidylinositol 3-kinase / protein kinase B ( PI3K / Akt ) signaling pathway .

6m2细胞转化与肌醇磷脂代谢相关性研究

The Relationship Between the Turnover of Inositol Phospholipids and 6m 2 Cell Transformation

T淋巴细胞内三磷酸肌醇传递conA诱导的增殖信号

Con A induced proliferative signal transduction mediated by ip_3 in T cells

目的探讨G蛋白-肌醇磷脂途径基因表达改变在原发性高血压病理机制中的作用。

Objective To investigate the changes of G protein-inositol phosphates pathway-related genes and evaluate the role of such changes in the pathogenesis of essential hypertension .

在清醒自由活动状态的大鼠上，肌醇磷酸酶抑制剂氯化锂、M受体激动剂氧化震颤素和N受体激动剂烟碱均可剂量依赖性地降低体温；

In conscious rats , inositol phosphatase inhibitor lithium chloride , muscarinic receptor agonist oxotremorine and nicotinic receptor agonist nicotine produced hypothermia in a dose dependent manner .

2,4-D和GA3对绿豆黄化幼苗肌醇磷脂代谢的影响

Effects of 2 , 4-D and GA_3 on Phosphoinositide Metabolism in Dark-Grown Phaseolus radiatus Seedlings

~（99m）Tc标记的肌醇六磷酸盐联合专利蓝检测早期宫颈癌前哨淋巴结

Sentinel lymph node detection in early cervical cancer with combination 99 mTc phytate and patent blue

PI3K（phosphoinositide3-kinase，磷脂酰肌醇-3-激酶）和AKT/PKB（蛋白激酶B）可以介导FAK的凋亡抑制效应；

Phosphoinositide 3-kinase and the AKT oncoprotein may mediate the anoikis-suppressing effects of focal adhesion kinase .

槲皮素对完整HL-60细胞中肌醇磷脂转换的抑制作用

Inhibitory effect of quercetin on the turnover of phosphoinositides in intact HL 60 cells

比较了在NT培养基中不同肌醇含量对原生质体的影响；

The effects of different inositol amounts in NT medium on the growth of protoplasts were compared .

吗啡长时程作用下小鼠脑组织磷酸肌醇含量和PKC活性的变化

Effects of chronic morphine treatment on inositol phosphates contents and PKC activity in mouse brain

目的促红细胞生成素（EPO）通过磷酸酰肌醇-3-激酶/蛋白激酶B（PI3-K/Akt）途径诱导血管生成和抑制细胞凋亡。

Objectives Erythropoietin ( EPO ) induces angiogenesis and inhibits apoptosis through the phosphatidylinositol-3-kinase / Akt ( PI3-K / Akt ) pathway .

本文简要介绍心肌细胞内皮素激活蛋白激酶C、丝裂原激活蛋白激酶、磷酸肌醇3'羟基激酶/蛋白激酶B等信号通路的调节作用。

This article briefly reviews the regulative effects of these signaling pathways including protein kinase C , mitogen activated protein kinase , and phosphoinositide 3 ' - OH kinase / protein kinase B.