缺血再灌注损伤

在CPB手术前使用阿魏酸钠可有效的抑制全身性炎症反应，减轻肺组织缺血再灌注损伤，有利于肺功能的保护。

SF could obviously reduce the activation of systemic inflammatory reaction , prevent lung IRI and has some good protective effects on the pulmonary function during and after CPB .

缺血再灌注损伤（IRI）是一种重要的病理生理现象。

Ischemia-reperfusion injury ( IRI ) is an important phenomenon of pathophysiology .

核因子-κB在急性脑缺血再灌注损伤中的作用及地塞米松的影响

Cerebral ischemia-reperfusion stimulated nf - κ b activation and the effect of dexamethasone in mice

脂质体前列腺素E1保护肝脏缺血再灌注损伤的实验研究

Experiment of Liposomal Prostaglandin E_1 Protects Against Hepatic Ischemia Injury

辅酶Q（10）与供心缺血再灌注损伤

Coenzyme Q_ ( 10 ) and Ischemia-reperfusion Injury of Donor Heart

目的探讨胸段硬膜外阻滞（TEA）对缺血再灌注损伤心肌的保护作用。

Objective : To explore the protective effects of thoracic epidural anesthesia ( TEA ) aganist myocardial ischemia-reperfusion injury .

结论丹参可能通过上调Bcl-2蛋白表达，下调Bax蛋白表达，对大鼠移植胰腺缺血再灌注损伤起保护作用。

Conclusion SM can decrease pancreatic ischemia-reperfusion injuries by up-regulating Bcl-2 and down-regulating Bax .

异氟烷预处理对缺血再灌注损伤鼠脑组织中Bcl-2和Bax的mRNA表达的影响

Effect of isoflurane preconditioning on Bcl-2 and Bax mRNA expression after ischemia / reperfusion in gerbil brain

背景：肠缺血再灌注损伤（Ischemiaandreperfusioninjury，简称I/R）是外科实践中常见的组织器官损伤之一。

BACKGROUND : Intestinal ischemia and reperfusion injury ( I / R ) is one of common tissue lesions in surgery practice .

肝脏缺血再灌注损伤（Hepaticischemiareperfusioninjury，HIR）是指肝脏缺血后重获血流灌注或氧供后，肝脏损伤会在缺血缺氧损伤的基础上进一步加重。

Background : Hepatic ischemia reperfusion ( HIR ) injury is a common acute disease of surgery .

肾缺血再灌注损伤的病理机制非常复杂，研究资料表明，iNOS的过表达参与病理生理过程。

Some studies indicates that iNOS expression increases in renal ischemia-reperfusion injury .

Caspase-3抑制剂对大鼠心肌缺血再灌注损伤后梗死范围的影响

Effects of caspase-3 inhibitor on infarct size of rats with myocardial reperfusion injury

目的观察异丙酚对大鼠胃缺血再灌注损伤（gastricischemia-ReperfusionInjury，GI-RI）的影响。

Objective To investigate the effect of propofol on gastric ischemia-reperfusion injury ( GI-RI ) .

目的：研究中华镜蛇毒F组分对脑缺血再灌注损伤的保护作用。

Objective : study the protective effect of fraction F of Naja naja atra venom on cerebral ischemia reperfusion injury in gerbils .

C-myc基因在供体小肠缺血再灌注损伤中的作用

The role of C-myc gene in the ischemic reperfusion injury of donor intestine

ICAM-1在移植心脏缺血再灌注损伤中的表达及意义

The Expression and Significance of ICAM-1 on Myocardial Reperfusion Injury after Cardiac Transplantation in Rats

三七总皂苷对大鼠脑缺血再灌注损伤血清IL-8的影响

Effect of Panax Notoginseng Saponins on the Content of IL-8 in Serum After Cerebral Ischemia-Reperfusion in rat

丹酚酸B对大鼠心肌缺血再灌注损伤内皮素及TXA2/PGI2系统的影响

Effect of salvianolic acid B on endothelin release and TXA2 / PGI_2 system in myocardial ischemia reperfusion injury in rats

目的观察异丙酚对离体大鼠全心缺血再灌注损伤心肌c-fos基因表达的影响，探讨其对缺血再灌注损伤心肌保护作用机制。

Objective To study the protective effects of propofol on isolated rat heart during global myocardial ischemia-reperfusion .

结论：iNOS可能在视网膜缺血再灌注损伤中发挥重要作用。

Conclusion : iNOS may play a very important role in ischemia-reperfusion injury in the rat retina .

人参、三七提取物与牛磺酸对兔心肌缺血再灌注损伤中ET、CGRP影响的实验研究

Experimental Study on Panax Ginseng and Panax Notoginseng Extraction plus Taurine in Protecting Myocardial Ischemia Reperfusion Injury from ET and CGRP

westernblot和明胶酶谱分析结果证明，白藜芦醇预治疗可降低脑缺血再灌注损伤所诱导的MMP-9蛋白的表达及其活性水平。

Results of Western blot and zymogram indicated that resveratrol could reduce the elevated levels of MMP-9 expression and activity induced by cerebral ischemia reperfusion .

目的探讨bcl-2基因对供体小肠缺血再灌注损伤的调控作用及机制。

Objective To investigate the effect of the regulation of the bcl-2 gene on the ischemia reperfusion injury of donor intestine and the mechanism .

结论：在再灌注前，及时给予依达拉奉可减少MDA的产生，减轻心肌缺血再灌注损伤。

Conclusion : Before reperfusion , MCI-186 can decrease the production of MDA in order to reduce myocardium ischemic reperfusion injury if supplied in time .

结论：川芎嗪能减少大鼠脂质过氧化，改善ATPase功能，减轻海马结构缺血再灌注损伤。

Conclusion TMP can reduce lipid peroxidation , improve ATPase activity and decrease neuronal damage in the IR injury of hippocampal structure .

Caspase-3在大鼠心肌缺血再灌注损伤中的作用及药物干预研究

The Study of the Role of Caspase-3 on Apoptosis During Myocardial Ischemia Reperfusion in Rats and the Effect of Ac-DEVD-CHO

大鼠合并缺血再灌注损伤肝大部切除后Ki-67、cyClinD1及TNF-α的表达

Expressions of ki-67 、 cyclin D1 and tnf - α in rats after major hepatectomy with ischemia reperfusion injury

结论：Epo可提高SOD，降低IL-6对肾缺血再灌注损伤具有保护作用。

Conclusion : EPO has protective effects on renal I / R injury in rats , because it can increase SOD and decrease IL-6 .

目的观察重组链激酶与尿激酶治疗急性心肌梗死（AMI）的疗效、副作用及缺血再灌注损伤。

Objective : To observe the therapeutic effect , side effects and ischemia-reperfusion injury of the thrombolytic therapy treated with recombinant streptokinase or with urokinase .

结论：EPO预处理可显著降低未成熟心肌缺血再灌注损伤，具有良好的心肌保护作用。

Conclusion : EPO pretreatment can significantly reduce the myocardial ischemia reperfusion injury , achieve better immature myocardial protective effect .